Paramedic ยท Module 2 of 5

ACLS & Cardiac Emergencies

Advanced Cardiac Life Support algorithms, STEMI recognition, cardioversion, and the full cardiac medication toolkit. Know these cold โ€” you'll use them on every serious cardiac call.

โ† All Modules 1Airway/RSI 2ACLS 3IV/Meds 4Trauma 5Medical Final Exam โ†’

ACLS Cardiac Arrest Algorithm

ACLS builds on BLS. The rhythm determines the treatment pathway. First: confirm arrest, start CPR, attach monitor/defibrillator.

Shockable Rhythms: VF / Pulseless VT

Non-Shockable Rhythms: PEA / Asystole

The H's and T's โ€” Reversible Causes of Cardiac Arrest

H's

  • Hypovolemia โ†’ fluids
  • Hypoxia โ†’ oxygenate
  • Hโบ (acidosis) โ†’ ventilate, bicarb
  • Hypo/Hyperkalemia โ†’ calcium/bicarb
  • Hypothermia โ†’ rewarm

T's

  • Tension pneumothorax โ†’ needle decompression
  • Tamponade โ†’ pericardiocentesis (hospital)
  • Toxins โ†’ specific antidotes
  • Thrombosis pulmonary (PE) โ†’ lytics
  • Thrombosis coronary (MI) โ†’ PCI

STEMI Recognition on 12-Lead ECG

In ERLC, STEMI recognition means knowing which territory is affected so you can communicate it and route to the right facility. ST elevation โ‰ฅ1mm in 2 contiguous leads in the same territory = STEMI until proven otherwise.

TerritoryLeads with ST ElevationArtery (IRL)Clinical Notes
InferiorII, III, aVFRCA (85%)Most common. Check for RV involvement (V4R). Nitro with caution โ€” can cause severe hypotension in RV MI.
AnteriorV1โ€“V4LADLargest territory. Highest mortality. May need pacing for complete heart block.
LateralI, aVL, V5โ€“V6LCxOften accompanies inferior or anterior MI.
PosteriorST depression V1โ€“V3, tall R waveRCA/LCxMirror image pattern โ€” depression in anterior leads = posterior elevation. Reciprocal changes.
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STEMI Transport

Identify STEMI โ†’ activate the cath lab early via radio/phone โ†’ transport directly to PCI-capable center (bypass non-PCI ED if within reason). Every minute of delay costs muscle. Door-to-balloon time goal is <90 minutes.

Cardioversion vs. Defibrillation

DefibrillationSynchronized Cardioversion
WhenPulseless VF or pVTUnstable tachyarrhythmia WITH a pulse (SVT, AF, VT with pulse)
SynchronizationUnsynchronized โ€” fires immediatelySynchronized โ€” fires on the R wave
Energy (biphasic)200J (or device-specific)SVT: 50โ€“100J start. VT with pulse: 100J start. AF: 120โ€“200J
SedationNot needed (patient is in arrest)Sedate if conscious (midazolam 2โ€“5 mg IV)
RiskConverts VF โ†’ shockable rhythm clearedMust be synchronized โ€” unsynchronized shock during T-wave can cause VF

Cardiac Arrest Medications

DrugDoseIndicationKey Points
Epinephrine1 mg IV/IO q3โ€“5minAll cardiac arrest rhythmsFirst drug in arrest. Alpha effects vasoconstrict โ†’ increases coronary perfusion pressure.
Amiodarone300 mg IV push, repeat 150 mgRefractory VF/pVT after 3rd shockBroadest antiarrhythmic. Hypotension common with IV push.
Lidocaine1โ€“1.5 mg/kg IVAlternative to amiodaroneUse if amiodarone unavailable. May repeat 0.5โ€“0.75 mg/kg q5โ€“10min, max 3 mg/kg.
Adenosine6 mg rapid IV push, flush; 12 mg if neededStable narrow-complex SVTHalf-life <10 seconds. Must be given rapid push with 20 mL saline flush. Antecubital or above.
Atropine0.5 mg IV q3โ€“5min, max 3 mgSymptomatic bradycardiaNot effective in complete heart block. Not used in arrest per current ACLS.

Transcutaneous Pacing

Indicated for: symptomatic bradycardia not responding to atropine, complete heart block, second-degree type II block. Requires the patient to have some cardiac activity (not for asystole).

1 mgEpinephrine dose (arrest)
300 mgAmiodarone first dose (VF)
6 mgAdenosine first dose (SVT)
3โ€“5 minEpinephrine interval